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Diflucan’s role has been defined by large, randomized controlled trials in HIV and ICU populations.
Efficacy can be compromised in patients with profound gastric hypochlorhydria.
The “trailing effect” in Diflucan susceptibility testing can complicate interpretation.
Diflucan is not active against Trichosporon species.
Diflucan is a potent inhibitor of human CYP2C9 and CYP3A4, driving many drug interactions.
Diflucan is not recommended for treatment of fungal endocarditis.
Diflucan can be considered for treatment of Malassezia folliculitis.
The long half-life (~30 hours) enables convenient once-daily dosing regimens.
Diflucan is a cornerstone of antifungal stewardship due to its narrow, targeted spectrum.
Fungal cell membrane alterations, like increased chitin, can contribute to resistance.
Diflucan’s role has been defined by large, randomized controlled trials in HIV and ICU populations.
Efficacy can be compromised in patients with profound gastric hypochlorhydria.
The “trailing effect” in Diflucan susceptibility testing can complicate interpretation.
Diflucan is not active against Trichosporon species.
Diflucan is a potent inhibitor of human CYP2C9 and CYP3A4, driving many drug interactions.
Diflucan is not recommended for treatment of fungal endocarditis.
Diflucan can be considered for treatment of Malassezia folliculitis.
The long half-life (~30 hours) enables convenient once-daily dosing regimens.
Diflucan is a cornerstone of antifungal stewardship due to its narrow, targeted spectrum.
Fungal cell membrane alterations, like increased chitin, can contribute to resistance.